wald chi-square Search Results


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CH Instruments asymptotic chi-square distribution of the wald statistic
Asymptotic Chi Square Distribution Of The Wald Statistic, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CH Instruments wald chi-squared test
Wald Chi Squared Test, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CH Instruments type ii wald chi-square tests
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Type Ii Wald Chi Square Tests, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CH Instruments wald chi-square test of parameter equalities
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Wald Chi Square Test Of Parameter Equalities, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CH Instruments wald log-linear chi-square test
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Wald Log Linear Chi Square Test, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CH Instruments nonlinear ecm neural network with 1 node in the first and second hidden layers
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Nonlinear Ecm Neural Network With 1 Node In The First And Second Hidden Layers, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CH Instruments type iii wald’s chi-square test
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Type Iii Wald’s Chi Square Test, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CH Instruments wald chi-square test for linearity
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Wald Chi Square Test For Linearity, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CH Instruments wald chi-square value minus degree of freedom
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Wald Chi Square Value Minus Degree Of Freedom, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CH Instruments wald chi square 103.6
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Wald Chi Square 103.6, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CH Instruments chi-square distribution wald test
Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II <t>Wald</t> Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice
Chi Square Distribution Wald Test, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CH Instruments maximum wald chi-square value
Effect of water level on: (A) queen survival, (B) queen wet mass, (C) queen water mass, (D) queen dry mass, and (E) number of brood (larvae, pupae, and workers) produced by single queens of the seed‐harvester ant Veromessor pergandei . Values are means + 1 SE . Within each panel, significant differences across moisture levels are indicated by the letters a‐b: a > b. Groupings are based on (A) a <t>Wald</t> chi‐square statistic (see text) or (B–E) a one‐way ANOVA followed by Duncan's multiple range test. Note that the one‐way ANOVA in panel D was not significant ( p = 0.08), while Duncan's multiple range test was significant ( p < 0.05). Initial sample size for each treatment is given in panel A; number of queens that survived to the end of the experiment (panels B–E) is given in panel B. ND = no data as no queens survived this treatment
Maximum Wald Chi Square Value, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II Wald Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice

Journal: Molecular Autism

Article Title: The activation of mGluR4 rescues parallel fiber synaptic transmission and LTP, motor learning and social behavior in a mouse model of Fragile X Syndrome

doi: 10.1186/s13229-023-00547-4

Figure Lengend Snippet: Enhanced SV docking of Fmr1 KO synapses and lack of an effect of isoproterenol. Isoproterenol (100 µM, 10 min) increases SV docking in WT ( a, c, d ) but not in Fmr1 KO ( e, g, h ) PF-PC synapses. Isoproterenol induces an increasing trend in the number of docked vesicles (0–10 nm) in WT ( a ) (n = 250/3 and 203/3 synapses/mice: P = 0.059) at the expense of SVs within 10–20 nm, which decreased in number (*** P < 0.001). These changes are absent in Fmr1 KO slices ( e ) (n = 238/4 and 224/4 synapses/mice, 0–10 nm: P = 0.997; 10–20 nm: P = 0.988). Note the increase in SV at 10–20 nm in Fmr1 KO control as opposed to WT control slices ( ## P < 0.01). c, d, g, h Electron microscopy of PF-PC synapses. (B,F) The effect of isoproterenol on the SV distribution: WT (0–5 nm: *** P < 0.001); Fmr1 KO (0–5 nm: P > 0.05). Fmr1 KO control vs WT control at 0–5 nm ( ## P < 0.01). ( i ), Active Zone (AZ) length of WT (n = 250) and Fmr1 KO (n = 238) synapses ( P > 0.05). The values represent the mean ± S.E.M. Scale bar in ( c, d, g, h ) 100 nm. Generalized Linear Mixed Models (GLMM) with genotype and treatment as fixed effects, and animal subject as a random effect. P-values for fixed effects and their interactions were obtained with type II Wald Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice

Article Snippet: P-values for fixed effects and their interactions were obtained with type II Wald Chi-Square tests and post-hoc tests were adjusted with the Tukey HSD method ( a, b, e, f ) and unpaired student’s t test in ( i ). n is the number of synapses, many synapses were analyzed per slice and several slices per mice

Techniques: Control, Electron Microscopy

Effect of water level on: (A) queen survival, (B) queen wet mass, (C) queen water mass, (D) queen dry mass, and (E) number of brood (larvae, pupae, and workers) produced by single queens of the seed‐harvester ant Veromessor pergandei . Values are means + 1 SE . Within each panel, significant differences across moisture levels are indicated by the letters a‐b: a > b. Groupings are based on (A) a Wald chi‐square statistic (see text) or (B–E) a one‐way ANOVA followed by Duncan's multiple range test. Note that the one‐way ANOVA in panel D was not significant ( p = 0.08), while Duncan's multiple range test was significant ( p < 0.05). Initial sample size for each treatment is given in panel A; number of queens that survived to the end of the experiment (panels B–E) is given in panel B. ND = no data as no queens survived this treatment

Journal: Ecology and Evolution

Article Title: Desiccation limits recruitment in the pleometrotic desert seed‐harvester ant Veromessor pergandei

doi: 10.1002/ece3.7039

Figure Lengend Snippet: Effect of water level on: (A) queen survival, (B) queen wet mass, (C) queen water mass, (D) queen dry mass, and (E) number of brood (larvae, pupae, and workers) produced by single queens of the seed‐harvester ant Veromessor pergandei . Values are means + 1 SE . Within each panel, significant differences across moisture levels are indicated by the letters a‐b: a > b. Groupings are based on (A) a Wald chi‐square statistic (see text) or (B–E) a one‐way ANOVA followed by Duncan's multiple range test. Note that the one‐way ANOVA in panel D was not significant ( p = 0.08), while Duncan's multiple range test was significant ( p < 0.05). Initial sample size for each treatment is given in panel A; number of queens that survived to the end of the experiment (panels B–E) is given in panel B. ND = no data as no queens survived this treatment

Article Snippet: Strength of the grouping effect can be assessed across water levels using the maximum Wald chi‐square value for comparisons across queen number within each water level.

Techniques: Produced

Percent survival for queens of the seed‐harvester ant Veromessor pergandei in the two‐factor bottle experiment (three queen numbers by three water levels). Significant differences across treatment cells are indicated by the letters a‐d: a > b > c > d. Groupings are based on the Wald chi‐square statistic (see text). Sample size per treatment cell; n = 120 queens for 0 and 5 mls water, n = 60 queens for 10 mls water

Journal: Ecology and Evolution

Article Title: Desiccation limits recruitment in the pleometrotic desert seed‐harvester ant Veromessor pergandei

doi: 10.1002/ece3.7039

Figure Lengend Snippet: Percent survival for queens of the seed‐harvester ant Veromessor pergandei in the two‐factor bottle experiment (three queen numbers by three water levels). Significant differences across treatment cells are indicated by the letters a‐d: a > b > c > d. Groupings are based on the Wald chi‐square statistic (see text). Sample size per treatment cell; n = 120 queens for 0 and 5 mls water, n = 60 queens for 10 mls water

Article Snippet: Strength of the grouping effect can be assessed across water levels using the maximum Wald chi‐square value for comparisons across queen number within each water level.

Techniques: